In a mouse model of diseases such as macular telangiectasia and retinal angiomatous proliferation, researchers have managed to prevent photoreceptor loss caused by abnormal growth of blood vessels without correcting the blood vessel defect.
In a mouse model of diseases such as macular telangiectasia and retinal angiomatous proliferation, researchers have managed to prevent photoreceptor loss caused by abnormal growth of blood vessels without correcting the blood vessel defect.