Mice with a defective mitochondrial protein called MCLK1 produce elevated amounts of reactive oxygen when young; that should spell disaster, yet according to a new study these mice actually age at a slower rate and live longer than normal mice.
Mice with a defective mitochondrial protein called MCLK1 produce elevated amounts of reactive oxygen when young; that should spell disaster, yet according to a new study these mice actually age at a slower rate and live longer than normal mice.